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Ycocalyx: acute deficits, but great potential. Cardiovasc Res. 2010;87:300?0. 35. Reitsma S, Slaaf

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Ycocalyx: acute deficits, but great potential. Cardiovasc Res. 2010;87:300?0. 35. Reitsma S, Slaaf DW, Vink H, van Zandvoort MA, oude Egbrink MG. The endothelial glycocalyx: composition, functions, and visualization. Pflugers Arch. 2007;454:345?9. 36. Senzolo M, Coppell J, Cholongitas E, Riddell A, Triantos CK, Perry D, et al. The effects of glycosaminoglycans on coagulation: a thromboelastographic study. Blood Coagul Fibrinolysis. 2007;18:227?6. 37. Rehm M, Bruegger D, Christ PubMed ID:https://www.ncbi.nlm.nih.gov/pubmed/12958591 F, Conzen P, Thiel M, Jacob M, et al. Shedding of the endothelial glycocalyx in patients undergoing major vascular surgery with global and regional ischemia. Circulation. 2007;116:1896?06. PubMed ID:https://www.ncbi.nlm.nih.gov/pubmed/4155310 38. Ostrowski SR, Johansson PI. Endothelial glycocalyx degradation induces endogenous heparinization in patients with severe injury and early traumatic coagulopathy. J Trauma Acute Care Surg. 2012;73:60?. 39. Scharbert G, Thaler U, Weilnbock C, Wetzel L, Kozek-Langenecker S. Heparininduced effects of prothrombin complex concentrates in thromboelastometry. Wien Klin Wochenschr. 2012;124:320?. tert-Butyl (7-bromoheptyl)carbamate 40. Renne T, Schmaier AH, Nickel KF, Blomback M, Maas C. In vivo roles of factor XII. Blood. 2012;120:4296?03. 41. Schlimp CJ, Cadamuro J, Solomon C, Redl H, Schochl H. The effect of fibrinogen concentrate and factor XIII on thromboelastometry in 33 diluted blood with albumin, gelatine, hydroxyethyl starch or saline in vitro. Blood Transfus. 2013;11:510?.Submit your next manuscript to BioMed Central and take full advantage of:?Convenient online submission ?Thorough peer review ?No space constraints or color figure charges ?Immediate publication on acceptance ?Inclusion in PubMed, CAS, Scopus and Google Scholar ?Research which is freely available for redistributionSubmit your manuscript at www.biomedcentral.com/submit
Tsurukiri et al. Scandinavian Journal of Trauma, Resuscitation and Emergency Medicine (2016) 24:13 DOI 10.1186/s13049-016-0205-ORIGINAL RESEARCHOpen AccessResuscitative endovascular balloon occlusion of the aorta for uncontrolled haemorrahgic shock as an adjunct to haemostatic procedures in the acute care settingJunya Tsurukiri1*, Itsurou Akamine1, Takao Sato1, Masatsugu Sakurai1, Eitaro Okumura1, Mariko Moriya1, Hiroshi Yamanaka1 and Shoichi OhtaAbstractBackground: Haemorrhagic shock is a major cause of death in the acute care setting. Since 2009, our emergency department has used intra-aortic balloon occlusion (IABO) catheters for resuscitative endovascular balloon occlusion of the aorta (REBOA). Methods: REBOA procedures were performed by one or two trained acute care physicians in the emergency room (ER) and intensive care unit (ICU). IABO catheters were positioned using ultrasonography. Collected data included clinical characteristics, haemorrhagic severity, blood cultures, metabolic values, blood transfusions, REBOA-related complications and mortality. Results: Subjects comprised 25 patients (trauma, n = 16; non-trauma, n = 9) with a median age of 69 years and a median shock index of 1.4. REBOA was achieved in 22 patients, but failed in three elderly trauma patients. Systolic blood 4-Bromo-5-nitro-1H-indazole pressure significantly increased after REBOA (107 vs. 71 mmHg, p < 0.01). Five trauma patients (20 ) died in ER, and mortality rates within 24 h and 60 days were 20 and 12 , respectively. No REBOA-related complications were encountered. The total occlusion time of REBOA was significantly lesser in survivors than that in non-survivors (52 vs. 97 min, p < 0.01). Significantly positive correla.

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